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Keith
Anderson is Research Lab Manager for the
Vascular Research Division and Senior Technician in the Gimbrone
Lab. Keith has been at the Brigham since 1994 after receiving an
MS in Biomedical Engineering from the University of Memphis and
a BS from Boston University. Keith's primary scientific focus is
on the fluid mechanical systems of the lab, including development
of devices for modeling fluid flow in the vasculature. |
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Guohao
Dai joined the Gimbrone laboratory as a postdoctoral fellow
in 2002. He received his Ph.D in Medical Engineering from Health
Sciences and Technology (HST) Program of Harvard and MIT, where
he worked on the molecular mechanism of deep venous thrombosis and
designing of mechanical methods for DVT prophylaxis. He is currently
using flow patterns derived from human atherosclerosis-prone and
atherosclerosis-protected regions of the vascular tree to investigate
the endothelium phenotype in health and disease. |
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Jeanne-Marie
Kiely is a Research Associate in the Gimbrone laboratory. With
extensive previous experience in Cellular Immunology, she has been
in the Vascular Research Division since 1989. Here she has been
exploring the role(s) of E-selectin particularly its role as a marker
of endothelial activation. Jeanne received a MS degree from Rutgers
University and a BS from Bates College. |
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Yvonne
Ou is a second-year medical student in the Harvard-MIT HST Division
of Harvard Medical School and began a research assistantship in
the Gimbrone lab in March, 2000. Yvonne's primary interest is how
biomechanical forces and biochemical signals alter gene expression
and endothelial cell phenotype. |
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Saran
Vaughn joined the lab of Dr. Gimbrone in the winter of 2002
as a technical research assistant. She earned her B.S. degree in
Biology from the University of Massachusetts at Amherst. Saran previously
worked at Wyeth Biopharma, where she assisted in the purification
and production of bone morphogenic protein. She has joined our group
with an interest in the study of how hemodynamic forces, such as
shear stress modulates endothelial structure and function. |